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Examination of Tetracycline pills system reveals a decrease in heart rate and an increase in blood pressure.

In the future, the stage of the height of the disease develops, lasting 8-12 days. In this period, remitting high fever and the development of shock reactions are observed. When examining patients, there is a bright redness and puffiness of the face, neck, sometimes subicteric (slight icterus) of the sclera and skin. Hemorrhages appear on the mucous membranes, often a hemorrhagic rash. In severe cases of Sumycin, gastrointestinal bleeding, hemoptysis are observed.

Tracheobronchitis and bronchopneumonia are often detected.

Often determined by an increase in the liver and spleen, as well as polyadenitis. Urinary excretion in patients is usually reduced. Depending on the severity of toxic and hemorrhagic syndromes, obliterated, mild, moderate and severe forms of the disease are distinguished. The latter are characterized by massive hemorrhagic syndrome, encephalitis, coma, renal failure and early death of patients (on the 3-5th day of illness).

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When examining blood, there is a decrease in the level of leukocytes, an increase in the number of lymphocytes, an almost complete absence of neutrophils, and a decrease in the level of platelets. A decrease in the level of prothrombin, fibrinogen and other blood coagulation factors is naturally detected.and. Often determined by the presence in the urine of albumin, blood, cylinders.

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The disease is recognized on the basis of Tetracycline data, clinical examination and laboratory tests. Specific diagnosis consists in the isolation of the virus in the first 3-12 days of the disease and antibodies that neutralize the virus at the end of the 3rd week of the disease.

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Specific therapy for Argentine hemorrhagic fever has not been developed. Symptomatic therapy is usually carried out, aimed at eliminating shock, intoxication. A blood transfusion is performed. A positive effect is given by intravenous administration of convalescent plasma to patients. The introduction of immune plasma significantly reduces mortality (from 16 to 1%), in an experiment on monkeys, the effectiveness of ribavirin (virazole) has been shown.

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Carry out pathogenetic therapy (rehydration, hemodialysis, etc.). Mortality ranges from 6 to 20%. Prevention of Argentine hemorrhagic fever.

Candid#1 is used to prevent adults at high risk of contracting Argentine hemorrhagic fever.

In 1985, the Argentine virologist Dr. Julio Barrera-Oro created a vaccine against AHF - Candid # 1. The vaccine was manufactured at the Salk Institute in the USA and has been available in Argentina since 1990.

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On August 29, 2006, the Maiztegui Institute received a certificate for vaccine production in Argentina. The vaccination plan has not yet been laid out, but the 2007 budget allows for 390,000 doses to be made, in Argentina it costs 8 pesos each (about $2.6 or €2). The institute has the capacity to produce the 5 million doses annually needed to vaccinate the entire population of tetracycline areas.

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Argentine hemorrhagic fever is a natural focal zoonosis from the group of South American hemorrhagic fevers, caused by an RNA-containing Junin virus. The leading clinical manifestations are hemorrhagic syndrome with bleeding of various localization, severe intoxication. Diagnosis is based on the detection of the pathogen itself and specific markers in the blood serum. Etiotropic treatment consists in the introduction of immune plasma. Good results are shown by the appointment of ribavirin.

Taking Sumycin into account the developed disorders, symptomatic therapy is prescribed. Causes Pathogenesis Symptoms of Argentine HF Complications Diagnosis Treatment of Argentine HF Prognosis and prevention Prices for treatment.

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The focus of Junin hemorrhagic fever is the humid ecosystem of the pampas of Argentina. Outbreaks of the disease occur annually from February to June with a peak in May. Most often, men get sick, the infection is more common among the rural population.

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